Skip to main content
. Author manuscript; available in PMC: 2007 Feb 13.
Published in final edited form as: J Biol Chem. 2006 Aug 7;281(39):28802–28810. doi: 10.1074/jbc.M603627200

Fig. 2.

Fig. 2

TAK1 is essential for osmotic stress-induced JNK but not p38.

(A) Schematic representation of TAK1 deletion.

(B) MEF cells and TAK1Δ/Δ were subjected to osmotic stress (0.5 M NaCl) for 10 or 30 min. Cell extracts were subjected to immunoblotting with anti-phospho JNK (top panel) and anti-phospho p38 (third panel). The amounts of endogenous JNK, p38, and TAK1 are shown in the second, fourth, and bottom panels, respectively. IB, immunoblotting.

(C) Mouse keratinocytes and TAK1Δ/Δ keratinocytes were subjected to osmotic stress (0.5 M NaCl) for 10 or 30 min. Cell extracts were subjected to immunoblotting with anti-phospho JNK (top panel) and anti-phospho p38 (third panel). The amounts of endogenous JNK, p38, and TAK1 are shown in the second, fourth, and bottom panels, respectively. IB, immunoblotting.