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. 2015 Sep 17;10(9):e0137826. doi: 10.1371/journal.pone.0137826

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© 2015 Gómez-Vicente et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Fig 3 — (A) Representative examples of filtered OP traces from scotopic ERGs recorded before (thin traces) and after (bold traces) NMDA-induced retinal damage, in vehicle- (upper graph) or TUDCA-treated (lower graph) rats, in response to a 1 cd·s/m² stimulus (arrow). (B) Amplitude (mean ± SEM) of maximum OPs before retinal damage (pre-lesion, n = 11), and after NMDA-induced lesion in rats either treated with vehicle (NMDA, n = 5) or TUDCA (NMDA+TUDCA, n = 6). Asterisks indicate statistical significance (ANOVA, Bonferroni’s test) for pre-lesion vs. NMDA, and NMDA vs. NMDA+TUDCA groups, **P<0.01. No significant differences were found when comparing pre-lesion vs. NMDA+TUDCA groups.