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Small molecule inhibition of maternal anti-brain autoantibodies. A. Anti-brain autoantibodies, which cross the placenta during gestation, can bind to fetal brain proteins and potentially inhibit important mechanisms in neurodevelopment. B. Small molecules that mimic the protein antigens can be used to compete with the endogenous antigens expressed in the fetal brain and neutralize the maternal autoantibodies to prevent antibody-mediated damage.
