Table 1.
Effects of whisker lesions of row C on TCA clustering in the cortex
|
Age at lesion |
Genotype |
Class I |
Class II |
Class III |
|---|---|---|---|---|
| P0-P1 | C3H (n = 9) | 89% | 11% | 0% |
| MA0A-K0 (n = 8) | 88% | 13% | 0% | |
| P2-P3 | C3H (n = 13) | 46% | 23% | 31% |
| MA0A-K0 (n = 14) | 31% | 46% | 23% | |
| P4-P5 | C3H (n = 19) | 0% | 0% | 100% |
|
|
MA0A-K0 (n = 13) |
0% |
0% |
100% |
Mice of the C3H and MA0A-K0 genotypes were lesioned at P0-P1, P2-P3, or P4-P5. The effects on barrel emergence were evaluated with 5-HTT or C0 staining at P9, P10, or P12. For each experimental group, cases were classified according to the degree of barrel segregation into three classes. Class I cases have a complete fusion of row C, class II cases have a partial fusion of row C, and class III cases have distinct barrel patterns in row C. Percentages of cases belonging to each class are provided for each experimental group.