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. Author manuscript; available in PMC: 2011 Apr 1.
Published in final edited form as: FEBS J. 2010 Feb 10;277(7):1618–1638. doi: 10.1111/j.1742-4658.2010.07588.x

Table 1.

Effect of IFG tartrate on N370S and L444P GCase activity in lysates from Gaucher patient-derived fibroblasts and LCLs.

Cell ID Mutation Cell Type GCase Activity
Baseline nmol/mg/hour IFG tartrate - μM (% increase) n
6 20 60
DMN89.45 N370S F 4.0±0.3 35±2 95±1 115±3 3
GM07968 L444P F 0.2±0.03 15±10 30±10 17±7 3
GM00877 L444P F 1.0±0.1 25±5 30±4 17±7 3
GM10915 L444P F 3.0±0.04 25±5 30±4 16±7 3
GM08760 L444P F 2.3±0.1 20±10 20±10 15±5 3
GS0501 L444P L 3.1±0.2 227±23 251±15 146±28 3
GS0505 L444P L 0.4±0.1 220±18 232±27 124±13 3
GS0502 L444P L 0.8±0.2 120±14 150±15 134±19 6
GS0503 L444P L 0.7±0.1 141±14 146±24 124±13 6
GS0504 L444P L 0.7±0.2 129±28 152±34 129±32 6

GCase activity in cell lysates was determined after five-day incubation of Gaucher fibroblasts or LCLs with the indicated concentrations of IFG tartrate. The GCase activity in fibroblasts derived from three different healthy volunteers (CRL1509, CRL2076, and CRL2097) was 25±2, 30±0.9, and 16±1.0 nmol/mg protein/hour, respectively. The average GCase activity in LCLs derived from two different healthy volunteers (GM02184 and GM03201) was 15±4 nmol/mg protein/hour. All cell lines were homozygous for the specified GCase mutations. The data for each cell line have been normalized to the GCase activity in untreated cells and are presented as the mean±SEM. ‘F’, fibroblast; ‘L’, lymphoblastoid cell line.